Modalities working with massage

Chapter Seven Modalities working with massage



In this chapter a number of modalities that integrate well with massage therapy will be discussed. To support proficiency, practical examples and skill enhancement exercises are included. Let’s begin by reviewing concepts from previous chapters.



Massage and pain management


Let us summarize the application of massage to target pain.


The massage professional, as part of a health care team, can contribute valuable manual therapy in various pain conditions using direct tissue manipulation and reflex stimulation of the nervous system and the circulation. As a therapeutic intervention, massage may help reduce the need for pain medication, thus reducing the side effects of medication.


All medications, including over-the-counter medication available without a prescription, have some side effects. Obviously, with clients in extreme pain, the massage therapy must be monitored by a doctor or other appropriate health care professional. Most people experience pain in less severe forms occasionally throughout life. Massage may provide temporary symptomatic relief of moderate pain brought on by daily stress, replacing over-the-counter pain medications or reducing their use.


Acute pain and chronic pain are managed somewhat differently; therefore it is important to make the distinction between the two. Intervention for acute pain is less invasive and focuses on supporting a current healing process. Chronic pain is managed with either symptom relief or a more aggressive healing and rehabilitation approach that incorporates a therapeutic change process.


Various mechanisms influencing pain are affected during massage. The neurotransmitters that perpetuate and inhibit the pain response are affected by massage application. The neurochemical most recognized by clients is endorphin. Endorphins are part of a group of peptides that act as the body’s internal pain modulator – like morphine. Endorphins have become recognized as part of the ‘runner’s high’ phenomenon; actually, a combination of neurotransmitters and hormones work together to alter pain perception, both inhibiting it and/or enhancing it. Massage seems to alter the chemical interaction. The pain inhibiting chemicals influenced by massage are from the entire endorphin class, as well as serotonin, GABA, e-cannabinoids, and dopamine. The pain facilitating chemicals influenced by massage are adrenaline, noradrenaline, cortisol, and substance P. The research is still scant on just how this all works, but what we understand is sufficient for strategic development and justification of massage for pain modulation.


Massage also influences the nervous system, central and peripheral (somatic and autonomic). Application of massage that results in counterirritation and hyperstimulation analgesia functions by activating the gate control for transmission of pain signals.


Reducing mechanical pressure on peripheral somatic nerves by increasing pliability in the tissues modulates pain sensation. Stimulation of nociceptors in tissues can be reduced by massage. Massage can inhibit the proprioceptors. When this occurs, joint function and the muscle tension–length relationship normalizes, decreasing pain. Supporting parasympathetic dominance increases pain tolerance.


Reducing hydrostatic pressure of edema using lymphatic drain application reduces interstitial fluid and decreases pressure on pain receptors. Similar results occur when tissue density is reduced, using connective tissue methods to increase ground substance pliability or to reduce adhesion from random connective tissue fiber distribution.


Pain can also occur if circulation is not appropriate. Ischemic tissues are sensitized to pain. Massage exerts a powerful influence on blood movement. Both arterial and venous circulation are involved and massage can target normalization. Massage also has a compassionate and comforting quality that can increase pain tolerance.



Pain management massage strategies


Massage application targeted to pain management incorporates the following principles:



1. General full-body application with a rhythmic and slow approach as often as feasible with 45–60 minute durations.


Goal – Parasympathetic dominance with reduced cortisol.


2. Pressure depth is moderate to deep with compressive broad based application. No poking, frictioning, or application of pain-causing methods.


Goal – Serotonin and GABA support and reduced substance P and adrenaline.


3. Drag is slight unless connective tissue is being targeted. Drag is targeted to lymphatic drain and skin stimulation.


Goal – Reduce swelling and create counterirritation through skin stimulation.


4. Nodal points on the body that have a high neurovascular component are massaged with a sufficient depth of pressure to create a ‘good hurt’ sensation but not defensive guarding or withdrawal. These nodal points are the location of cutaneous nerves, trigger points, acupuncture points, reflexology points, etc. The foot, hands, and head, as well as along the spine, are excellent target locations.


Goal – Gate control response, endorphin and other pain-inhibiting chemical release.


5. Direction of massage varies, but deliberately targets fluid movement.


Goal – Improved circulation.


6. Mechanical force introduction of shear, bend, torsion, etc. are of an agitation quality to ‘stir’ the ground substance and not create inflammation.


Goal – Increased tissue pliability and reduced tissue density.


7. Mechanical force application of shear, bend, and torsion is used to address adhesion or fibrosis but needs to be specifically targeted and limited in duration.


Goal – Reduce localized nerve irritation or circulation reduction.


8. Muscle energy methods and lengthening are applied rhythmically, gently, and targeted to shortened muscles.


Goal – Reduce nerve and proprioceptive irritation and circulation inhibition.


9. Stretching to introduce tension force is applied slowly, without pain and targeted to shortened connective tissue.


Goal – Reduce nerve and proprioceptive irritation.


10. Massage therapists are focused, attentive, compassionate, but maintain appropriate boundaries.


Goal – Support entrainment, bioenergy normalization, and palliative care.


Now that the massage application and mechanisms have been described, what else can be done?





General adaptation sequence (see fig. 7.2)


Selye’s (1978) General Adaptation Syndrome describes a process in which the individual, with his/her unique inherited and acquired characteristics, is responding to multiple variable or constant adaptive demands, resulting in:







Therapeutic choices available are limited to those that:






Interacting causes


Very few pain conditions arise from a single cause. Symptoms may emerge from a background involving very different stressors, interacting with the unique genetic, biomechanical, biochemical, and psychosocial characteristics of the individual.


Clearly any person whose system is coping with multiple types, levels, and degrees of compensation and adaptation, is likely to be susceptible to provocation by a variety of further stress factors – ‘triggering’ events or influences – including, as examples, additional physical and/or psychological strain, or a change in atmospheric pressure (such as occurs before a thunder storm), or a draft from air conditioning, or an infection, or an allergic response, or…a host of other possibilities.



Or, are the many interacting stresses the ‘causes:’ the biochemical, psychosocial, biomechanical factors to which the local area of pain (common locations are head, neck, low back, and lower limb joints) and the person as a whole, are adapting? What has led to the stage where a single additional stress (the trigger) can exacerbate the pain? Much depends on just how long adaptive stressor demands have been operating – weeks, months, years, or a lifetime? Two factors are commonly found regardless of the cause of pain. If massage can interact in these areas it is likely that pain intensity can be reduced and symptoms managed.





The sensitization model


Bendtsen (2000) has described a process of central sensitization (facilitation) that occurs after prolonged bombardment of pain messages from pain receptors (nociceptors) in myofascial tissues. At its simplest this means that nerves have become hyperirritable, so that even minor stimuli, that would previously not have caused any discomfort, can lead to a great deal of pain. This does not mean that the person is imagining the pain, but that the sensations reaching the brain are interpreted as being far stronger than would be the case under ‘normal’ conditions, before sensitization.


The research supporting this model demonstrates the need to understand how, over time, a reversible problem may become entrenched and chronic.


Once they exist, areas of facilitation/sensitization appear to be capable of being irritated by stressors of all types – physical, chemical, or psychological – even if there is no direct or obvious impact on the sensitized area (Bendtsen & Ashina 2000).




Janda’s example of adaptation and facial pain


Janda (1982) describes a typical postural pattern, in an individual with TMJ problems, involving changes in upper trapezius, levator scapulae, scalenii, sternocleidomastoid, suprahyoid, lateral and medial pterygoid, masseter, and temporalis muscles. In this pattern (described below) all these muscles will show a tendency to tighten and/or to develop tendencies to spasm, tenderness, and the evolution of trigger points.


The postural pattern associated with TMJ dysfunction might therefore involve:



The message that can be drawn from these examples is that dysfunction patterns first need to be identified before they can be assessed for the role they might be playing in the person’s pain and restriction conditions, and certainly before these can be successfully and appropriately treated.


Additionally, general full body massage that does not overtask the adaptive process is the intervention of choice when these adaptive patterns predominate. Initially (and maybe never) it is unlikely that we will know what is causing what. Where the current pain is located is likely not the causal factor of the pain but instead related to the adaptive response. We must realize that adaptation is important for function in the midst of the onslaught of life. The adaption that has occurred has been the best the body could do. Without this appreciation, the massage therapist may inadvertently unsettle a productive compensation pattern and the client ends up in more pain. The lesson is to progress slowly and rely on the massage approach as presented in this text. As the massage continues for the individual it is imperative to gather information so that the general massage can be refined to best assist in pain management. Ongoing assessment needs to identify:


What’s short? What’s tight? What’s weak? What’s loose? What’s affecting neighboring structures negatively? What’s causing these changes, and what can be done about it without aggravating the situation?



What’s to be done?


In any given case your role is to try to identify what can be done to help ease the current pain symptom (see previous chapters, and also Box 7.1), as well as what might be done, or what the person might do, to reduce the likelihood of further symptomatic episodes.



In these examples, using a biomechanical model of care, incorporating massage and appropriate soft tissue modalities (see Box 7.1) and rehabilitation strategies (posture, breathing, etc.) should certainly help to alter/improve the soft tissue status, enhance circulation and lymphatic drainage, and, if appropriate, assist in the deactivating of trigger points that may be contributing to pain.



A broader model of care


The biomechanical model outlined in Box 7.1 is one way of managing such problems. Other proposed models for effective care of musculoskeletal dysfunction incorporate somatic, as well as behavioral features. For example, Langevin & Sherman (2006) have described a model in which a broader therapeutic approach to musculoskeletal dysfunction in general can be understood.


This is an ‘integrative mechanistic’ model that addresses both behavioral and structural aspects, as well as pain psychology, postural control, and neuroplasticity.


This model emphasizes the need, in many instances, for multidisciplinary treatment protocols, possibly including direct biomechanical/manual approaches including massage, movement reeducation, psychosocial interventions, and where necessary, pharmacological and/or nutritional treatment methods and modalities that meet the particular needs of the individual.


In this way long-term preventive approaches might also include:




Jun 19, 2016 | Posted by in PAIN MEDICINE | Comments Off on Modalities working with massage

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