Headache and Orofacial Pain

Introduction


Headache and orofacial disorders comprise a heterogenous group of disorders of varying prevalence (Box 9.1). Despite being extremely common, headache and orofacial disorders are under-recognised and under-diagnosed and can result in considerable pain and disability. The second edition of the International Classification of Headache Disorders (ICHD-II) lists more than100 headache and facial pain conditions (see Further reading). These can be further classified by cause (Figure 9.1). In this chapter, strategies for diagnosis are outlined and headache and facial pain that is often misdiagnosed or inadequately managed by non-specialists is the focus.







Box 9.1 Causes of Headache and Facial Pain



Prevalence >10%

Hangover

Tension-type headache

Fever

Metabolic disorders

Sinusitis

Migraine

Temporomandibular disorders

Persistent idiopathic facial pain


Prevalence >1% ≤10%

Medication overuse headache

Idiopathic stabbing headache


Prevalence ≤1%

Giant cell arteritis

Cranial neuralgias

Trigeminal

Glossopharyngeal

Occipital

Trigeminal autonomic cephalalgias (TACs)

Cluster headache

Chronic Paroxysmal hemicrania

Short-lasting unilateral neuralgiform headache attacks with conjunctival injection and tearing (SUNCT), Short-lasting unilateral neuralgiform headache attacks with autonomic features (SUNA)

Trigeminal neuropathy

Post-herpetic neuralgia






Figure 9.1 Classification of headache and orofacial pain (ICHD-II code). TACs: trigeminal autonomic cephalalgias.

9.1

History


The history is a crucial step in diagnosis (Box 9.2). Patients often present with more than one type of pain, so a separate history is required for each. In the emergency setting, there may not be time to take a full history. The first task is to exclude conditions requiring more urgent intervention by eliciting any warning features (Table 9.1).







Box 9.2 Approach to the History



1. How many different headache types does the patient experience?

Separate histories are necessary for each. It is reasonable to concentrate on the most bothersome to the patient but others should always attract some enquiry in case they are clinically important.

2. Time questions

a. Why consulting now?

b. How recent in onset?

c. How frequent, and what temporal pattern (especially distinguishing between episodic and daily or unremitting)?

d. How long lasting?


3. Character questions

a. Intensity of pain

b. Nature and quality of pain

c. Site and spread of pain

d. Associated symptoms

4. Cause questions

a. Predisposing and/or trigger factors

b. Aggravating and/or relieving factors

c. Family history of similar headache

5. Response to headache questions

a. What does the patient do during the headache?

b. How much is activity (function) limited or prevented?

c. What medication has been and is used, and in what manner?

6. State of health between attacks

a. Completely well, or residual or persisting symptoms?

b. Concerns, anxieties, fears about recurrent attacks, and/or their cause

Based on: MacGregor, EA, Steiner, TJ & Davies, PTG (2010) Guidelines for All Healthcare Professionals in the Diagnosis and Management of Migraine, Tension-Type, Cluster and Medication-Overuse Headache. Available at: http://www.bash.org.uk


Table 9.1 Warning features in the history





















































Symptoms Possible diagnosis Investigation
Thunderclap headache (intense headache with abrupt or ‘explosive’ onset) Subarachnoid haemorrhage CT without contrast
LP (if CT normal or unavailable)
Headache with atypical aura (duration >1 hour, or including motor weakness) TIA
Ischaemic stroke
CT with contrast/MRI
Fever Meningitis LP


Blood cultures
History of HIV or syphilis Encephalopathy Antibody testing


LP


MRI
History of cancer Secondary brain metastases CT with contrast/MRI
New onset headache over age 50 Giant cell arteritis ESR/CRP


Temporal artery biopsy

Glaucoma Ophthalmological review
Progressive headache worsening over weeks or months Intracranial space-occupying lesion CT with contrast/MRI
Symptoms of raised intracranial pressure

  • Drowsiness
  • Postural related headache
  • Vomiting
New onset seizures
Cognitive or personality changes
Progressive neurological deficit

  • Weakness
  • Sensory loss
  • Dysphasia
  • Ataxia

Progressive sensory loss over the face and/or intraorally Intracranial space-occupying lesion Neurophysiological testing, CT or MRI

aCT: computed tomography; CRP: C-reactive protein; ESR: erythrocyte sedimentation rate; LP: lumbar puncture; MRI: magnetic resonance imaging; TIA: transient ischaemic attack


Examination


The examination will reflect the suspected diagnosis, particularly in the case of local disease of the oral cavity. A good light and expertise in examining the oral tissues is necessary. The head and neck should be carefully examined. Assess tenderness over the temporal arteries as well tenderness in the scalp, neck muscles and muscles of mastication. Examination of patients with temporomandibular disorders (TMD) involves the application of pressure to a variety of specific anatomical sites to see if they are tender (trigger points). The range of movements of the mandible is then evaluated in vertical opening, protrusive and lateral positions. A brief but thorough neurological examination, especially of the cranial nerves, should be undertaken, particularly if the diagnosis is uncertain or intracranial pathology is suspected. Fundoscopy is mandatory in all patients presenting with headache. Examination of the cranial nerves is particularly important for orofacial pain. General examination is indicated if headaches are considered secondary to systemic disease.


Investigations


There is no indication for investigations of primary headaches and they should be undertaken only if a secondary headache is suspected. If autoimmune disorders are suspected then blood tests may be indicated. Non-contrast computed tomography (CT) with axial and coronal sections is indicated for sinusitis, rhinogenic causes of pain or if malignancy is suspected. Magnetic resonance imaging (MRI) is useful to evaluate internal derangement in TMD. MRI and/or magnetic resonance angiography (MRA) can evaluate intracranial pathology. Special thin cut MRIs of the posterior fossa are needed to identify trigeminal nerve root compressions.


Management Issues


Patients’ beliefs are fundamental and must be clarified before treatment is initiated (Box 9.3). Severity of disease, impact on quality of life, insight and chronicity will impact on adherence to treatment as well as how the family views the condition (Box 9.4). The approach must, therefore, be a biopsychosocial one.


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Jun 14, 2016 | Posted by in PAIN MEDICINE | Comments Off on Headache and Orofacial Pain

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