9faf5978d6e6d55b92bf8309a1d}/ID(AB1-M91)” href=”javascript:void(0)” title=”Hgb” onmouseover=”window.status=this.title; return true;” onmouseout=”window.status=”; return true;” onclick=”get_content(event,’AB1-M91′); return false;” target=”right”>Hgb molecule is capable of binding four oxygen molecules. Although oxygen dissolves in blood, binding oxygen to Hgb found in RBCs allows the blood to carry 70 times as much oxygen. In arterial blood, about 98.5% of the oxygen is bound to Hgb and only 1.5% is dissolved within the blood. V being specific to the monitoring techniques that should be used to assess a patient’s oxygenation, ventilation, circulation, and body temperature. These monitors are discussed in roughly the same order as the standards they fulfill.
provide values for other states of Hgb, such as carbhemoglobin, carboxyhemoglobin, methemoglobin, and sulfhemoglobin, as well as the total amount of Hgb. Co-oximeters are commonly available with blood gas machines; however, pulse co-oximeters are available for bedside use as well.
Light: Ambient light normally affects both the saturation reading and the pulse rate. Shield the sensor with a surgical towel, drapes, etc. or change the sensor site.
Motion: Motion of the probe interferes with readings. This can occur when the probe is jostled (e.g., surgical personnel leaning agaisub>2 monitoring is to confirm the placement
of an endotracheal tube into the trachea. If the tube is placed in the trachea and the patient is ventilated, and there is sufficient cardiac output to deliver CO2 to the lungs and there is sufficient gas exchange between the blood and the alveoli, the CO2 monitor will register CO2. If the endotracheal tube is placed in the esophagus, the CO2 monitor will not register sustained CO2 (Fig. 33.3). Although there may be some carbon dioxide in the esophagus and stomach, the concentration is normally low and will rapidly be depleted with ventilation of the stomach.
▪ FIGURE 33.2 A-E: Normal capnogram. A,B: Baseline represents continued inhalation (value should be zero) or lack of gas movement. B,C: Expiratory upstroke (sharp rise from baseline represents the beginning of exhalation and consists of a mixture of air and alveolar gas. C,D: Expiratory plateau (continued exhalation of alveolar gas, should be straight or nearly straight). D: End-tidal concentration (value at the end of exhalation); D,E: Inspiration begins (sharp downstroke as fresh gapter 32″ onmouseover=”window.status=this.title; return true;” onmouseout=”window.status=”; return true;” onclick=”get_content(event,’B01745940-DA4-C32′); return false;” target=”right”>Chapter 32. In this chapter, we focus on the practical application of that technology in anesthesia. The role of capnography within the ASA standards is to help ensure a patient is adequately ventilated during all anesthetics. Continuous readings of exhaled carbon dioxide (CO2) can instantly communicate changes in the status of the patient and the anesthesia machine.
Although the numerical readings from a capnometer will satisfy the basic requirements, a capnograph with a continuous waveform is preferred. The shape of the waveform can yield additional diagnostic information about how a patient is being ventilated.
The “normal” capnogram is a waveform that represents the varying CO2 level throughout the breath cycle over time (Fig. 33.2A-E).
Measured exhaled CO2 is a function of CO2 production by the body, delivery of the CO2-containing blood to the lungs, gas exchange in the alveoli, and ventilation of the lungs to pick up CO2 from the alveoli and eliminate it to the outside. One of the most important functions of CO
Machine problems leading to decreased ventilation (leaks, obstructions, depleted CO2 absorber, etc.)
▪ FIGURE 33.4 Rising ETCO2 levels as detected on a capnogram. (Adapted from Capnography Self-Study Guide. Rev. 1. Smiths Medical; 2008. Used by permission.)
Decrs.title; return true;” onmouseout=”window.status=”; return true;”>Fig. 33.3). Although there may be some carbon dioxide in the esophagus and stomach, the concentration is normally low and will rapidly be depleted with ventilation of the stomach.
Below is a discussion of the differential diagnosis of changes in measured CO2 values or waveforms. Increased CO2: An increase in the level of End-Tidal Carbon dioxide (ETCO2) from previous levels can result from either an increase in the patient’s production of CO2 or a decrease in ventilation (Fig. 33.4):
Increased metabolic rate (rising body temperature from blankets or external warming devices, thyrotoxicosis, malignant hyperthermia, etc.)
Increased cardiac output
Chemicals or metabolic products that have been administered to the patient that are converted to CO2 (bicarbonate, lactate, CO2 gas embolus, etc.)
Decreased respiratory rate or tidal volume
Decreased gas exchange with eventual rising CO2 blood levels (pulmonary failure, chronic obstructive pulmonary disease [COPD], bronchospasm, etc.)
Machine problems leading to decreased ventilation (leaks, obstructions, depleted CO2 absorber, etc.)
▪ FIGURE 33.4 Rising ETCO2 levels as detected on a capnogram. (Adapted from Capnography Self-Study Guide. Rev. 1. Smiths Medical; 2008. Used by permission.)
Decreased CO2: A decrease in the level of ETCO2 from previous levels can result from a change in the body’s production of CO2 (rare), the delivery of CO2 to the lungs, or an increase in ventilation (Fig. 33.5):
Decreased metabolic rate or decreased core body temperature
Decreased cardiac output
Decreased pulmonary blood flow (pulmonary embolus)
Increased tidal volume or respiratory rate
Partial disconnect of CO2-monitoring tubing
▪ FIGURE 33.3 ETCO2 levels rapidly diminish or are not present with an esophageal intubation. (Adapted from Capnography Self-Study Guide. Rev. 1. Smiths Medical; 2008. Used by permission.)
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